By : Robyn Chuter
We’re nearly two years into the COVID-19 debacle, and nothing that governments anywhere in the world have done to “stop the spread” and “end the pandemic” has worked. Nothing. Nada. Big fat zero.
If you have any remaining doubt about that, I urge you to read Dr Paul Elias Alexander’s outstanding summary of the damning evidence, More Than 400 Studies on the Failure of Compulsory Covid Interventions. The title kind of gives it away, but let me give you a quick summary:
SARS-CoV-2 is a highly contagious respiratory virus that is spread primarily through airborne transmission, which means that sanitising your hands, disinfecting surfaces and maintaining an arbitrary distance from other people is pointless biosecurity theatre – pointless, that is, in preventing the spread of the virus, but that was never the real intention anyway.
The primary route of entrance of SARS-CoV-2 is via inhalation of tiny droplet nuclei, or aerosols, that are less than 5 micrometres (5 μm) in diameter. That is, we breathe it into our nostrils. (And by the way, cloth masks increase the dose of those tiny aerosols that end up in your nose.)
Luckily for us, we’ve co-evolved with viruses, so our bodies know a thing or two about how to handle them. The secretory immunoglobulin A system, or sIgA, is our first line of defence against both respiratory and gastrointestinal viral infections.
sIgA is the most prevalent type of antibody produced in the human body. It is produced by immune system cells known as lymphocytes, that live just under the mucous membranes that line our respiratory and gastrointestinal tracts (the respiratory and gut mucosa).
As soon as a viral antigen (a snippet of protein that the immune system recognises as foreign to the human body) comes in contact with the respiratory mucosa, lymphocytes release sIgA which is ejected through the mucous membrane onto the mucosal surface, where it can prevent viruses from binding to cells and thus infecting them:
From ‘The COVID vaccines were designed to fail’
At the same time as they’re firing their antiviral missiles through the respiratory mucosa, these crack troop lymphocytes send for reinforcements by signalling other immune system cells that live in the central lymphoid organs in the bronchial and intestinal mucosa to also pump out sIgA. Thus, a call-to-arms propagates through every mucous membrane-lined organ in the human body, along with military intelligence and a specific battle plan on how to recognise and defend against the viral intruder.
If secretory IgA levels are high enough, the virus will be stopped in its tracks, before it gets the chance to enter enough cells to generate an infection. If they aren’t, and if our innate immune mechanisms don’t manage to fend off the attack, the virus will replicate and will make its way down into the lower respiratory tract from where it can infect the lungs. This is the pattern that SARS-CoV-2 follows.
sIgA levels are lowered by suboptimal vitamin D levels, obesity, and possibly by zinc deficiency – so well done to those public health geniuses who designed COVID-19 policies that restricted going outdoors into the vitamin D-generating sunshine, and resulted in 35 per cent of Australians gaining weight, mostly because of increased junk food consumption.
Unlike the johnny-on-the-spot secretory IgA which mops up respiratory viruses at their point of entry into the body, the novel injections marketed as “COVID-19 vaccines” only generate the classes of antibodies known as immunoglobulin G (IgG) and circulating immunoglobulin A (IgA).
As the name of the latter class implies, these are antibodies which circulate in the blood, searching for virus that has breached the mucosal defences and broken through into the bloodstream (which is easy to do once the virus has invaded the highly-vascularised lungs) in order to try to invade internal organs.
Sufficiently high levels of these antibodies do help to prevent the organ damage that characterises severe COVID-19, which is why studies such as those I cited in my previous article, The COVID-19 vaccine treadmill, show an initial decrease in the risk of serious illness, hospitalisation and death related to COVID-19, which tapers off dramatically after around 6 months (sooner in males, frail elderly people and those with comorbidities) as vaccine-generated antibody levels plummet.
However, these blood-borne antibodies cannot neutralise SARS-CoV-2 in the respiratory mucosa because they don’t reach it. And this is why COVID-19 injections don’t stop infection with SARS-CoV-2, don’t stop viral replication in the nasopharynx (nose and throat), and don’t stop transmission of the virus.
I, and many others, have said it before, over and over and over and over and over and over and over and over again: The current crop of “COVID-19 vaccines” cannot possibly end the pandemic because they are incapable of generating herd immunity.
What they do, unfortunately, is to
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